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Distinct ligands, such as 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and the tryptophan metabolite, 6-formylindolo[3,2-b]carbazole (FICZ), induce unique conformational changes in AhR, affecting its interaction with the exenobiotic response element (XRE), the core DNA sequence regulating gene transcription in the AhR signaling pathway

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